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プロフィール詳細
プロジェクトを作成
★★★★★
☆☆☆☆☆
Dr. Caterina D.に依頼
Italy

Medical Writer and Scientific Vsual Communicator

プロフィール概要
専門分野
サービス
Writing Medical Writing, General Proofreading & Editing
Data & AI Data Visualization
職務経験

Associate Research Scientist

Yale University

3月 2020 - 12月 2022

Scientific Illustrator Intern

Mount Sinai Hospital

2月 2022 - 5月 2022

Postdoctoral Associate

Yale University

3月 2016 - 3月 2021

PhD student

Vita-Salute San Raffaele University

1月 2010 - 4月 2015

Master student

KU Leuven

3月 2008 - 2月 2009

学歴

PhD (DRI)

Vita-Salute San Raffaele University

1月 2010 - 4月 2015

Master Degree

University of Bologna

9月 2006 - 3月 2009

Bachelor Degree

University of Bologna

9月 2002 - 6月 2006

認定資格
  • Certificate of study in Scientific Illustration

    Scientific Illustration Distance Program (SIDP)

    1月 2022 - 現在

出版物
JOURNAL ARTICLE
Caterina Di Pietro, Ann M. Haberman, Brett D. Lindenbach, Peter C. Smith, Emanuela M. Bruscia, Heather Allore, Brent Vander Wyk, Antariksh Tyagi, Caroline Zeiss (2024). Prior Influenza Infection Mitigates SARS-CoV-2 Disease in Syrian Hamsters . Viruses.
Hasan H. Öz, Ee-Chun Cheng, Caterina Di Pietro, Toma Tebaldi, Giulia Biancon, Caroline Zeiss, Ping-Xia Zhang, Pamela H. Huang, Sofia S. Esquibies, Clemente J. Britto, et al. (2022). Recruited monocytes/macrophages drive pulmonary neutrophilic inflammation and irreversible lung tissue remodeling in cystic fibrosis . Cell Reports.
Caterina Di Pietro, Hasan H. Öz, Ping-xia Zhang, Ee-chun Cheng, Valentino Martis, Tracey L. Bonfield, Thomas J. Kelley, Ronald Jubin, Abraham Abuchowski, Diane S. Krause, et al. (2022). Recruitment of monocytes primed to express heme oxygenase-1 ameliorates pathological lung inflammation in cystic fibrosis . Experimental & Molecular Medicine.
Caterina Di Pietro, Hasan H. Öz, Thomas S. Murray, Emanuela M. Bruscia (2020). Targeting the Heme Oxygenase 1/Carbon Monoxide Pathway to Resolve Lung Hyper-Inflammation and Restore a Regulated Immune Response in Cystic Fibrosis . Frontiers in Pharmacology.
Caterina Di Pietro, Ping-xia Zhang, Timothy K. O'Rourke, Jr., Thomas S. Murray, Lin Wang, Clemente J. Britto, Jonathan L. Koff, Diane S. Krause, Marie E. Egan, Emanuela M. Bruscia (2017). Ezrin links CFTR to TLR4 signaling to orchestrate anti-bacterial immune response in macrophages . Scientific Reports.
Caterina Di Pietro, Lorena De Giorgi, Ilaria Cosorich, Chiara Sorini, Maya Fedeli, Marika Falcone (2016). MicroRNA-133b Regulation of Th-POK Expression and Dendritic Cell Signals Affect NKT17 Cell Differentiation in the Thymus . The Journal of Immunology.
Jayashree Dolpady, Chiara Sorini, Caterina Di Pietro, Ilaria Cosorich, Roberto Ferrarese, Diego Saita, Massimo Clementi, Filippo Canducci, Marika Falcone (2016). Oral Probiotic VSL#3 Prevents Autoimmune Diabetes by Modulating Microbiota and Promoting Indoleamine 2,3-Dioxygenase-Enriched Tolerogenic Intestinal Environment . Journal of Diabetes Research.
Caterina, Di Pietro (2014). The role of invariant NKT cells in organ-specific autoimmunity . Frontiers in Bioscience.
Jeroen Declercq, Anujith Kumar, Conny Gysemans, Caterina Di Pietro, Anica Schraenen, Marie Chintinne, Katleen Lemaire, Leentje Van Lommel, Marc Van De Casteele, Harry Heimberg, et al. (2011). <i>Pdx1</i>- and <i>Ngn3-Cre</i>-Mediated <i>PLAG1</i> Expression in the Pancreas Leads to Endocrine Hormone Imbalances That Affect Glucose Metabolism . Cell Transplantation.
Simone Caielli, Cristina Conforti-Andreoni, Caterina Di Pietro, Vera Usuelli, Ester Badami, Maria Luisa Malosio, Marika Falcone (2010). On/Off TLR Signaling Decides Proinflammatory or Tolerogenic Dendritic Cell Maturation upon CD1d-Mediated Interaction with Invariant NKT Cells . The Journal of Immunology.
Jeroen Declercq, Anujith Kumar, Janna A. Van Diepen, Irene O.C.M. Vroegrijk, Conny Gysemans, Caterina Di Pietro, Peter J. Voshol, Chantal Mathieu, Nadine Ectors, Wim J.M. Van de Ven, et al. (2010). Increased β-Cell Mass by Islet Transplantation and <i>PLAG1</i> Overexpression Causes Hyperinsulinemic Normoglycemia and Hepatic Insulin Resistance in Mice . Diabetes.