プロフィール詳細

プロジェクトを作成

★★★★★

☆☆☆☆☆

★★★★★

☆☆☆☆☆

Dr. Helina M.に依頼

United Kingdom

Freelance Editor, Medical Writer and Microbiology Consultant with 13+ years of experience

プロフィール概要

Infectious Disease specialist, Editor-in-Chief and freelance medical writer

専門分野

• ★ Microbiology
• ★ Infectious Disease
• ☆ Medical Sciences

サービス

• Writing
• Research
• Consulting
• Data & AI
• Product Development

Writing Technical Writing, Newswriting

Research Market Research, User Research, Fact Checking, Gap Analysis, Gray Literature Search, Systematic Literature Review, Secondary Data Collection

Consulting Scientific and Technical Consulting

Data & AI Statistical Analysis, Data Visualization, Data Processing

Product Development Product Evaluation

職務経験

Queen's University Belfast

- 現在

Research scientist / Research fellow

University College Dublin

3月 2019 - 現在

Postdoctoral Research Fellow

Queen's University Belfast

7月 2016 - 現在

Research assistant

Baylor College of Medicine

5月 2011 - 9月 2011

Intern

Baylor College of Medicine

6月 2010 - 9月 2010

学歴

Clinical Microbiology

University College London, University of London

9月 2012 - 6月 2016

BSc (Hons) Biological Sciences

Edinburgh Napier University

10月 2008 - 6月 2012

認定資格

• 認定資格の詳細は未入力です。

出版物

JOURNAL ARTICLE

Helina Marshall, Maria Fernandes(2021). Early‐career researchers shaping publishing strategy . Learned Publishing. 34. (4). Microsoft.AspNetCore.Mvc.Localization.LocalizedHtmlString 675--678. Wiley

Early‐career researchers shaping publishing strategy @article{1d65ea41c19b4f80979004f76423d31a, title = "Early‐career researchers shaping publishing strategy", abstract = "If society publishers listen to and work with their early‐career members, we have an opportunity to reclaim our place as trusted, valued players in the research communication space. Early‐career researchers can provide fresh insights into publishing strategy, creating opportunities for publishers to learn about what the next generation of our customers truly value. Pairing early‐career researchers with experienced mentors has proven a successful strategy for building an innovative, engaged Editorial Board.", author = "Helina Marshall and Maria Fernandes", year = "2021", month = mar, day = "24", doi = "10.1002/leap.1383", language = "English", journal = "Learned Publishing", issn = "0953-1513", publisher = "Wiley Online Library", } . Learned Publishing.

Helina Marshall, Julie P. Meneely, Brian Quinn, Yueju Zhao, Paula Bourke, Brendan F. Gilmore, Guangtao Zhang, Christopher T. Elliott(2020). Novel decontamination approaches and their potential application for post-harvest aflatoxin control . Trends in Food Science & Technology. 106. Microsoft.AspNetCore.Mvc.Localization.LocalizedHtmlString 489--496. Elsevier {BV}

Helina Marshall, Daniel Storey, Matthew R. Parsek, Alan McNally, Mia Åstrand, Joana sa-Pessoa Graca Santos, Isabel Rodriguez-Escudero, Bronagh Elmore, Leyre Palacios, Laura Hobley, et al.(2020). Klebsiella pneumoniae type VI secretion system-mediated microbial competition is PhoPQ controlled and reactive oxygen species dependent . PLOS Pathogens. 16. (3). Microsoft.AspNetCore.Mvc.Localization.LocalizedHtmlString e1007969. Public Library of Science ({PLoS})

Klebsiella pneumoniae type VI secretion system-mediated microbial competition is PhoPQ controlled and reactive oxygen species dependent @article{0d9ea03131ff411fa971cf0c901d98a9, title = "Klebsiella pneumoniae type VI secretion system-mediated microbial competition is PhoPQ controlled and reactive oxygen species dependent", abstract = "Klebsiella pneumoniae is recognized as an urgent threat to human health due to the increasing isolation of multidrug resistant strains. Hypervirulent strains are a major concern due to their ability to cause life-threating infections in healthy hosts. The type VI secretion system (T6SS) is widely implicated in microbial antagonism, and it mediates interactions with host eukaryotic cells in some cases. In silico search for genes orthologous to T6SS component genes and T6SS effector genes across 700 K. pneumoniae genomes shows extensive diversity in T6SS genes across the K. pneumoniae species. Temperature, oxygen tension, pH, osmolarity, iron levels, and NaCl regulate the expression of the T6SS encoded by a hypervirulent K. pneumoniae strain. Polymyxins and human defensin 3 also increase the activity of the T6SS. A screen for regulators governing T6SS uncover the correlation between the transcription of the T6SS and the ability to kill E. coli prey. Whereas H-NS represses the T6SS, PhoPQ, PmrAB, Hfq, Fur, RpoS and RpoN positively regulate the T6SS. K. pneumoniae T6SS mediates intra and inter species bacterial competition. This antagonism is only evident when the prey possesses an active T6SS. The PhoPQ two component system governs the activation of K. pneumoniae T6SS in bacterial competitions. Mechanistically, PhoQ periplasmic domain, and the acid patch within, is essential to activate K. pneumoniae T6SS. Klebsiella T6SS also mediates anti-fungal competition. We have delineated the contribution of each of the individual VgrGs in microbial competition and identified VgrG4 as a T6SS effector. The DUF2345 domain of VgrG4 is sufficient to intoxicate bacteria and yeast. ROS generation mediates the antibacterial effects of VgrG4, and the antitoxin Sel1E protects against the toxic activity of VgrG4. Our findings provide a better understanding of the regulation of the T6SS in bacterial competitions, and place ROS as an early event in microbial competition.", author = "Daniel Storey and Alan McNally and Mia {\AA}strand and {Sa-Pessoa Graca Santos}, Joana and Isabel Rodriguez-Escudero and Bronagh Elmore and Leyre Palacios and Helina Marshall and Laura Hobley and Maria Molina and Cid, {Victor J} and Salminen, {Tiina A} and Bengoechea, {Jose A}", year = "2020", month = mar, day = "19", doi = "10.1371/journal.ppat.1007969", language = "English", volume = "16", journal = "PLoS Pathogens", issn = "1553-7366", publisher = "Public Library of Science", number = "3", } . PLoS Pathogens.

(2020). In Vivo Relationship between the Nano-Biomechanical Properties of Streptococcal Polysaccharide Capsules and Virulence Phenotype . ACS nano.

In Vivo Relationship between the Nano Biomechanical Properties of Streptococcal Polysaccharide Capsules and Virulence Phenotype @article{b84da1c4cbec4896bd3f954ddb8cedc1, title = "In Vivo Relationship between the Nano Biomechanical Properties of Streptococcal Polysaccharide Capsules and Virulence Phenotype", abstract = "In common with many bacterial pathogens, Streptococcus pneumoniae has a polysaccharide capsule which facilitates immune evasion and determines virulence. Recent data have shown that the closely related Streptococcus mitis also expresses polysaccharide capsules including those with an identical chemical structure to S. pneumoniae capsular serotypes. We utilized atomic force microscopy (AFM) techniques to investigate the biophysical properties of S. mitis and S. pneumoniae strains expressing the same capsular serotypes that might relate to differences in virulence potential. When comparing S. mitis and S. pneumoniae strains with identical capsule serotypes, S. mitis strains were susceptible to neutrophil killing, and electron microscopy and AFM demonstrated significant morphological differences. Force–volume mapping using AFM showed distinct force–curve profiles for the center and edge areas of encapsulated streptococcal strains. This “edge effect” was not observed in unencapsulated bacteria and therefore was a direct representation of the mechanical properties of the bacterial capsule. When two strains of S. mitis and S. pneumoniae expressed an identical capsular serotype, they presented similar biomechanical characteristics. This infers a potential relationship between capsule biochemistry and nanomechanics, independent of bacterial strain. Overall, this study demonstrates that it is possible to investigate reproducibly the mechanistic, structural, and mechanical properties of both the capsule and the body of individual living bacterial cells and relate the data to virulence phenotypes. We have demonstrated that using nanomechanics to investigate individual bacterial cells we can now begin to identify the surface properties bacterial pathogens require to avoid host-mediated immunity.", author = "Helina Marshall and Sebastian Aguayo and Mogens Kilian and Fernanda Petersen and Laurent Bozec and Jeremy Brown", year = "2019", month = dec, day = "19", doi = "10.1021/acsnano.9b08631", language = "English", journal = "ACS Nano", issn = "1936-0851", publisher = "American Chemical Society", } . ACS Nano.

Helina Marshall, Norman K. Fry, Tasha Mellins-Cohen(2019). In praise of preprints . Journal of Medical Microbiology. 68. (4). Microsoft.AspNetCore.Mvc.Localization.LocalizedHtmlString 503--505. Microbiology Society

Helina Marshall, Norman K. Fry, Tasha Mellins-Cohen(2019). In praise of preprints . Access Microbiology. 1. (2). Microbiology Society

Fry NK, Marshall H, Mellins-Cohen T(2019). In praise of preprints . Microbial genomics.

Fry NK, Marshall H, Mellins-Cohen T(2019). In praise of preprints . Microbiology (Reading, England).

Fry NK, Marshall H, Mellins-Cohen T(2019). In praise of preprints . The Journal of general virology.

Fry NK, Marshall H, Mellins-Cohen T(2019). In praise of preprints . Journal of medical microbiology.

Fry NK, Marshall H, Mellins-Cohen T(2019). In praise of preprints . International journal of systematic and evolutionary microbiology.

Fry, N.K., Marshall, H., Tasha Mellins-Cohen(2019). In praise of preprints . Microbiology (United Kingdom). 165. (5). Microsoft.AspNetCore.Mvc.Localization.LocalizedHtmlString 489-491.

Fry, N.K., Marshall, H., Mellins-Cohen, T.(2019). In praise of preprints . International Journal of Systematic and Evolutionary Microbiology. 69. (7). Microsoft.AspNetCore.Mvc.Localization.LocalizedHtmlString 1841-1843.

Fry, N.K., Marshall, H., Mellins-Cohen, T.(2019). In praise of preprints . Journal of General Virology. 100. (5). Microsoft.AspNetCore.Mvc.Localization.LocalizedHtmlString 733-735.

Fry, N.K., Marshall, H., Mellins-Cohen, T.(2019). In praise of preprints . Journal of Medical Microbiology. 68. (4). Microsoft.AspNetCore.Mvc.Localization.LocalizedHtmlString 503-505.

Early Adhesion of Candida albicans onto Dental Acrylic Surfaces @article{d5251d6a802343f792b1cb4beab306f1, title = "Early Adhesion of Candida albicans onto Dental Acrylic Surfaces", abstract = "Denture-associated stomatitis is a common candidal infection that may give rise to painful oral symptoms, as well as be a reservoir for infection at other sites of the body. As poly (methyl methacrylate) (PMMA) remains the main material employed in the fabrication of dentures, the aim of this research was to evaluate the adhesion of Candida albicans cells onto PMMA surfaces by employing an atomic force microscopy (AFM) single-cell force spectroscopy (SCFS) technique. For experiments, tipless AFM cantilevers were functionalized with PMMA microspheres and probed against C. albicans cells immobilized onto biopolymer-coated substrates. Both a laboratory strain and a clinical isolate of C. albicans were used for SCFS experiments. Scanning electron microscopy (SEM) and AFM imaging of C. albicans confirmed the polymorphic behavior of both strains, which was dependent on growth culture conditions. AFM force-spectroscopy results showed that the adhesion of C. albicans to PMMA is morphology dependent, as hyphal tubes had increased adhesion compared with yeast cells ( P < 0.05). C. albicans budding mother cells were found to be nonadherent, which contrasts with the increased adhesion observed in the tube region. Comparison between strains demonstrated increased adhesion forces for a clinical isolate compared with the lab strain. The clinical isolate also had increased survival in blood and reduced sensitivity to complement opsonization, providing additional evidence of strain-dependent differences in Candida-host interactions that may affect virulence. In conclusion, PMMA-modified AFM probes have shown to be a reliable technique to characterize the adhesion of C. albicans to acrylic surfaces.", keywords = "Acrylic Resins, Biofilms, Candida albicans, Cell Adhesion, Dental Materials, Microscopy, Atomic Force, Microscopy, Electron, Scanning, Polymethyl Methacrylate, Spectrum Analysis, Surface Properties, Journal Article", author = "S Aguayo and H Marshall and J Pratten and D Bradshaw and Brown, {J S} and Porter, {S R} and D Spratt and L Bozec", year = "2017", month = jul, day = "1", doi = "10.1177/0022034517706354", language = "English", volume = "96", pages = "917--923", journal = "Journal of Dental Research", issn = "0022-0345", publisher = "AMER ASSOC DENTAL RESEARCH", number = "8", } . Journal of Dental Research.

Naturally Acquired Human Immunity to Pneumococcus Is Dependent on Antibody to Protein Antigens @article{276da8528f2245d88c2206697a6c0eab, title = "Naturally Acquired Human Immunity to Pneumococcus Is Dependent on Antibody to Protein Antigens", abstract = "There are errors in the penultimate sentence of the “S. pneumonia target antigens vary partially between individuals and with age” section in the Results. The citations to Fig 3D, Fig 3E and Fig 3F should refer to Fig 4D, Fig 4E and Fig 4F. The sentence should read: In general, mean anti-protein antigen responses were slightly lower for the aged subjects (Fig 4D), with the most marked differences being for PspC (Fig 4E) and PcpA (Fig 4F).", keywords = "Published Erratum", author = "Robert Wilson and Cohen, {Jonathan M} and Mark Reglinski and Jose, {Ricardo J} and Chan, {Win Yan} and Helina Marshall and {de Vogel}, Corn{\'e} and Stephen Gordon and David Goldblatt and Petersen, {Fernanda C} and Helen Baxendale and Brown, {Jeremy S}", year = "2017", month = jan, day = "30", doi = "10.1371/journal.ppat.1006259", language = "English", volume = "13", pages = "e1006259", journal = "PLoS Pathogens", issn = "1553-7366", publisher = "Public Library of Science", number = "3", } . PLoS Pathogens.

Wilson, R., Cohen, J.M., Reglinski, M., Jose, R.J., Chan, W.Y., Marshall, H., de Vogel, C., Gordon, S., Goldblatt, D., Petersen, F.C., et al.(2017). Erratum: Correction: Naturally Acquired Human Immunity to Pneumococcus Is Dependent on Antibody to Protein Antigens (PLoS pathogens (2017) 13 1 (e1006137)) . PLoS pathogens. 13. (3). Microsoft.AspNetCore.Mvc.Localization.LocalizedHtmlString e1006259.

Aguayo, S., Marshall, H., Pratten, J., Bradshaw, D., Brown, J.S., Porter, S.R., Spratt, D., Bozec, L.(2017). Early Adhesion of Candida albicans onto Dental Acrylic Surfaces . Journal of Dental Research. 96. (8). Microsoft.AspNetCore.Mvc.Localization.LocalizedHtmlString 917-923.

Wilson, R., Cohen, J.M., Reglinski, M., Jose, R.J., Chan, W.Y., Marshall, H., de Vogel, C., Gordon, S., Goldblatt, D., Petersen, F.C., et al.(2017). Naturally Acquired Human Immunity to Pneumococcus Is Dependent on Antibody to Protein Antigens . PLoS Pathogens. 13. (1).

Importance of bacterial replication and alveolar macrophage-independent clearance mechanisms during early lung infection with Streptococcus pneumoniae @article{932499c57f924cd985929610305b66ef, title = "Importance of bacterial replication and alveolar macrophage-independent clearance mechanisms during early lung infection with Streptococcus pneumoniae", abstract = "Although the importance of alveolar macrophages for host immunity during early Streptococcus pneumoniae lung infection is well established, the contribution and relative importance of other innate immunity mechanisms and of bacterial factors are less clear. We have used a murine model of S. pneumoniae early lung infection with wild-type, unencapsulated, and para-amino benzoic acid auxotroph mutant TIGR4 strains to assess the effects of inoculum size, bacterial replication, capsule, and alveolar macrophage-dependent and -independent clearance mechanisms on bacterial persistence within the lungs. Alveolar macrophage-dependent and -independent (calculated indirectly) clearance half-lives and bacterial replication doubling times were estimated using a mathematical model. In this model, after infection with a high-dose inoculum of encapsulated S. pneumoniae, alveolar macrophage-independent clearance mechanisms were dominant, with a clearance half-life of 24 min compared to 135 min for alveolar macrophage-dependent clearance. In addition, after a high-dose inoculum, successful lung infection required rapid bacterial replication, with an estimated S. pneumoniae doubling time of 16 min. The capsule had wide effects on early lung clearance mechanisms, with reduced half-lives of 14 min for alveolar macrophage-independent and 31 min for alveolar macrophage-dependent clearance of unencapsulated bacteria. In contrast, with a lower-dose inoculum, the bacterial doubling time increased to 56 min and the S. pneumoniae alveolar macrophage-dependent clearance half-life improved to 42 min and was largely unaffected by the capsule. These data demonstrate the large effects of bacterial factors (inoculum size, the capsule, and rapid replication) and alveolar macrophage-independent clearance mechanisms during early lung infection with S. pneumoniae.", keywords = "4-Aminobenzoic Acid, Animals, Bacterial Capsules, Bacterial Load, Female, Half-Life, Immunity, Innate, Lung, Macrophages, Alveolar, Male, Mice, Mice, Inbred Strains, Models, Statistical, Mutation, Phagocytosis, Pneumonia, Pneumococcal, Severity of Illness Index, Streptococcus pneumoniae, Time Factors, Journal Article, Research Support, Non-U.S. Gov't", author = "Emilie Camberlein and Cohen, {Jonathan M} and Ricardo Jos{\'e} and Hyams, {Catherine J} and Robin Callard and Suneeta Chimalapati and Jose Yuste and Edwards, {Lindsey A} and Helina Marshall and {van Rooijen}, Nico and Mahdad Noursadeghi and Brown, {Jeremy S}", note = "Copyright {\textcopyright} 2015, American Society for Microbiology. All Rights Reserved.", year = "2015", month = mar, doi = "10.1128/IAI.02788-14", language = "English", volume = "83", pages = "1181--1189", journal = "Infection and Immunity", issn = "0019-9567", publisher = "American Society for Microbiology", number = "3", } . Infection and Immunity.

Camberlein, E., Cohen, J.M., Jos&#233;, R., Hyams, C.J., Callard, R., Chimalapati, S., Yuste, J., Edwards, L.A., Marshall, H., van Rooijen, N., et al.(2015). Importance of bacterial replication and alveolar macrophage-independent clearance mechanisms during early lung infection with Streptococcus pneumoniae . Infection and Immunity. 83. (3). Microsoft.AspNetCore.Mvc.Localization.LocalizedHtmlString 1181-1189.

Protective role of the capsule and impact of serotype 4 switching on Streptococcus mitis @article{07ca5f3bd254417a963d852f25ccf7f0, title = "Protective role of the capsule and impact of serotype 4 switching on Streptococcus mitis", abstract = "The polysaccharide capsule surrounding Streptococcus pneumoniae is essential for virulence. Recently, Streptococcus mitis, a human commensal and a close relative of S. pneumoniae, was also shown to have a capsule. In this study, the S. mitis type strain switched capsule by acquisition of the serotype 4 capsule locus of S. pneumoniae TIGR4, following induction of competence for natural transformation. Comparison of the wild type with the capsule-switching mutant and with a capsule deletion mutant showed that the capsule protected S. mitis against phagocytosis by RAW 264.7 macrophages. This effect was enhanced in the S. mitis strain expressing the S. pneumoniae capsule, which showed, in addition, increased resistance against early clearance in a mouse model of lung infection. Expression of both capsules also favored survival in human blood, and the effect was again more pronounced for the capsule-switching mutant. S. mitis survival in horse blood or in a mouse model of bacteremia was not significantly different between the wild type and the mutant strains. In all models, S. pneumoniae TIGR4 showed higher rates of survival than the S. mitis type strain or the capsule-switching mutant, except in the lung model, in which significant differences between S. pneumoniae TIGR4 and the capsule-switching mutant were not observed. Thus, we identified conditions that showed a protective function for the capsule in S. mitis. Under such conditions, S. mitis resistance to clearance could be enhanced by capsule switching to serotype 4, but it was enhanced to levels lower than those for the virulent strain S. pneumoniae TIGR4.", keywords = "Animals, Bacteremia, Bacterial Capsules, Cell Line, Disease Models, Animal, Female, Horses, Humans, Lung, Macrophages, Mice, Mice, Inbred BALB C, Phagocytosis, Respiratory Tract Infections, Serotyping, Streptococcal Infections, Streptococcus mitis, Streptococcus pneumoniae, Virulence, Journal Article, Research Support, Non-U.S. Gov't", author = "Rukke, {H{\aa}kon V} and Kalluru, {Raja Sab} and Urska Repnik and Alice Gerlini and Jos{\'e}, {Ricardo J} and Jimstan Periselneris and Helina Marshall and Gareth Griffiths and Oggioni, {Marco Rinaldo} and Brown, {Jeremy S} and Petersen, {Fernanda C}", note = "Copyright {\textcopyright} 2014, American Society for Microbiology. All Rights Reserved.", year = "2014", month = sep, doi = "10.1128/IAI.01840-14", language = "English", volume = "82", pages = "3790--3801", journal = "Infection and Immunity", issn = "0019-9567", publisher = "American Society for Microbiology", number = "9", } . Infection and Immunity.

Rukke, H.V., Kalluru, R.S., Repnik, U., Gerlini, A., Jos&#233;, R.J., Periselneris, J., Marshall, H., Griffiths, G., Oggioni, M.R., Brown, J.S., et al.(2014). Protective role of the capsule and impact of serotype 4 switching on Streptococcus mitis . Infection and Immunity. 82. (9). Microsoft.AspNetCore.Mvc.Localization.LocalizedHtmlString 3790-3801.

OTHER

Klebsiella pneumoniae type VI secretion system-mediated microbial competition is PhoPQ controlled and reactive oxygen species dependent @misc{1585322001254ef099df2fb415b6df94, title = "Klebsiella pneumoniae type VI secretion system-mediated microbial competition is PhoPQ controlled and reactive oxygen species dependent", abstract = "Klebsiella pneumoniae is recognized as an urgent threat to human health due to the increasing isolation of multidrug resistant strains. Hypervirulent strains are a major concern due to their ability to cause life-threating infections in healthy hosts. The type VI secretion system (T6SS) is widely implicated in microbial antagonism, and it mediates interactions with host eukaryotic cells in some cases. In silico search for genes orthologous to T6SS component genes and T6SS effector genes across 700 K. pneumoniae genomes shows extensive diversity in T6SS genes across the K. pneumoniae species. Temperature, oxygen tension, pH, osmolarity, iron levels, and NaCl regulate the expression of the T6SS encoded by a hypervirulent K. pneumoniae strain. Polymyxins and human defensin 3 also increase the activity of the T6SS. A screen for regulators governing T6SS uncover the correlation between the transcription of the T6SS and the ability to kill E. coli prey. Whereas H-NS represses the T6SS, PhoPQ, PmrAB, Hfq, Fur, RpoS and RpoN positively regulate the T6SS. K. pneumoniae T6SS mediates intra and inter species bacterial competition. This antagonism is only evident when the prey possess an active T6SS. The PhoPQ two component system governs the activation of K. pneumoniae T6SS in bacterial competitions. Mechanistically, PhoQ periplasmic domain, and the acid patch within, is essential to activate K. pneumoniae T6SS. Klebsiella T6SS also mediates anti-fungal competition. We have delineated the contribution of each of the individual VgrGs in microbial competition, and identified VgrG4 as a T6SS effector. Structurally, domain DUF2345 of VgrG4 is sufficient to intoxicate bacteria and yeast. ROS generation mediates the antibacterial effects of VgrG4, and the antitoxin Sel1E protects against the toxic activity of VgrG4. Our findings provide a better understanding of the regulation of the T6SS in bacterial competitions, and place ROS as an early event in microbial competition.", author = "Helina Marshall", year = "2019", month = jul, day = "10", language = "English", type = "Other", } .

Helina Marshall, Daniel Storey, Alan McNally, Mia &#197;strand, Joana S&#225;-Pessoa Graca Santos, Isabel Rodriguez-Escudero, Bronagh Elmore, Leyre Palacios, Laura Hobley, Maria Molina Martin, et al.(2019). Klebsiella pneumoniaetype VI secretion system-mediated microbial competition is PhoPQ controlled and reactive oxygen species dependent . Cold Spring Harbor Laboratory

In praise of preprints @article{3ca9f661ae0f4a33939ae20d797ba7c3, title = "In praise of preprints", abstract = "Tom Sheldon{\textquoteright}s article published in July 2018 entitled {\textquoteleft}Preprints could promote confusion and distortion{\textquoteright} generated some heated debate and responses in both social media and the scientific literature. Here we present some thoughts and views from the Microbiology Society, a not for-profit membership charity for scientists interested in microbes and the publisher of a number of international peer-reviewed journals.", keywords = "Editorial policy, Peer review, Preprints, Social media", author = "Fry, {Norman K.} and Helina Marshall and Tasha Mellins-Cohen", note = "Funding Information: Citable preprints allow authors to establish priority for the work they have done by providing a public record. This is so well accepted that most funding bodies, including UKRI [19], Wellcome [20] and the US National Institutes of Health [21], allow researchers to cite preprints in their grant applications. Copyright: Copyright 2019 Elsevier B.V., All rights reserved.", year = "2019", month = jul, doi = "10.1099/ijsem.0.003323", language = "English", volume = "69", pages = "1841--1843", journal = "International Journal of Systematic and Evolutionary Microbiology", issn = "1466-5026", publisher = "Society for General Microbiology", number = "7", } . International Journal of Systematic and Evolutionary Microbiology.

In praise of preprints @article{79162200f3da45eba935a4035376b1dd, title = "In praise of preprints", keywords = "Editorial policy, Peer review, Preprints, Social media", author = "Fry, {Norman K.} and Helina Marshall and {Tasha Mellins-Cohen}, Mellins-Cohen", note = "Funding Information: Citable preprints allow authors to establish priority for the work they have done by providing a public record. This is so well accepted that most funding bodies, including UKRI [19], Wellcome [20] and the US National Institutes of Health [21], allow researchers to cite preprints in their grant applications. Copyright: Copyright 2019 Elsevier B.V., All rights reserved.", year = "2019", month = may, day = "1", doi = "10.1099/mic.0.000785", language = "English", volume = "165", pages = "489--491", journal = "Microbiology (United Kingdom) ", issn = "1350-0872", publisher = "Society for General Microbiology", number = "5", } . Microbiology (United Kingdom).

Helina Marshall, Norman K. Fry, Tasha Mellins-Cohen(2019). In Praise of Pre-Prints . Microbial Genomics. 5. (4). Microbiology Society

In praise of preprints @article{e5a622add3cf48c6aa48427609d76845, title = "In praise of preprints", abstract = "Tom Sheldon{\textquoteright}s article published in July 2018 entitled {\textquoteleft}Preprints could promote confusion and distortion{\textquoteright} [1] generated some heated debate and responses in both social media and the scientific literature [2–4]. Here we present some thoughts and views from the Microbiology Society, a not-for-profit membership charity for scientists interested in microbes and the publisher of a number of international peer-reviewed journals.", keywords = "Editorial policy, Peer review, Preprints, Social media", author = "Fry, {Norman K.} and Helina Marshall and Tasha Mellins-Cohen", note = "Funding Information: Citable preprints allow authors to establish priority for the work they have done by providing a public record. This is so well accepted that most funding bodies, including UKRI [19], Wellcome [20] and the US National Institutes of Health [21], allow researchers to cite preprints in their grant applications. Copyright: Copyright 2019 Elsevier B.V., All rights reserved.", year = "2019", month = apr, day = "2", doi = "10.1099/jmm.0.000954", language = "English", volume = "68", pages = "503--505", journal = "Journal of Medical Microbiology", issn = "0022-2615", publisher = "Society for General Microbiology", number = "4", } . Journal of Medical Microbiology.

In praise of preprints @article{52b1516af7834cf18edfd62d40cd35c1, title = "In praise of preprints", keywords = "Editorial policy, Peer review, Preprints, Social media", author = "Fry, {Norman K.} and Helina Marshall and Tasha Mellins-Cohen", note = "Funding Information: Citable preprints allow authors to establish priority for the work they have done by providing a public record. This is so well accepted that most funding bodies, including UKRI [19], Wellcome [20] and the US National Institutes of Health [21], allow researchers to cite preprints in their grant applications. Copyright: Copyright 2019 Elsevier B.V., All rights reserved.", year = "2019", month = apr, day = "2", doi = "10.1099/jgv.0.001239", language = "English", volume = "100", pages = "733--735", journal = "Journal of General Virology", issn = "0022-1317", publisher = "Society for General Microbiology", number = "5", } . Journal of General Virology.