プロフィール詳細

プロジェクトを作成

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Dr. KE K.に依頼

United States

Scientific researcher with 10+ years of experience in osteoimmunology

プロフィール概要

My primary focus during my graduate study, was to identify the featuring pathophysiological role of various bioactive signaling molecules, as well as their application as the potential promising therapeutic strategy for the treatment of osteoporosis. During my post-doctoral training period, I was engaged in alternative projects, including investigation on interplay between the immune system and metabolic pathway in the context of rheumatoid arthritis or osteoarthritis. Those dynamic circumstances and experiences provide me with necessary skills for academic-oriented career, and those skills include the independent research skills, grant-based scientific writing, interprofessional teamwork ability, as well as adaptation to a more challengeable working environment.

専門分野

• ★ Metabolism & Metabolic Disease
• ★ Technical & Scientific Writing
• ☆ Immunoassay
• ☆ Oxidative Stress & Mitochondrial Function
• ☆ Molecular Biology
• ☆ Lipidomics
• ☆ Cell Signaling Pathways
• ☆ Stem Cell Research
• ☆ Data Analysis
• ☆ Paleontology
• ☆ Cell Aging
• ☆ Diabetes
• ☆ Obesity
• ☆ Osteoporosis
• ☆ Musculoskeletal Oncology
• ☆ Peer Review
• ☆ Systematic Literature Review

サービス

• Writing
• Research
• Data & AI
• Product Development

Writing Technical Writing

Research Market Research, Gap Analysis, Systematic Literature Review

Data & AI Statistical Analysis

Product Development Concept Development

職務経験

Visiting Researcher

Washington University in St.louis

8月 2017 - 6月 2021

Postdoctroral Researcher

Ulsan University

11月 2013 - 6月 2017

学歴

Doctor of Philosophy (Ph.D)

Ulsan University

9月 2008 - 8月 2013

認定資格

• 認定資格の詳細は未入力です。

出版物

JOURNAL ARTICLE

Sul OJ, Kim JE, Ke K, Suh JH, Choi HS(2021). Corrigendum to "Atherogenic diet-induced bone loss is primarily due to increased osteoclastogenesis in mice" [Journal of Nutritional Biochemistry 79C (2020) 108337] . The Journal of nutritional biochemistry.

Arra M, Swarnkar G, Ke K, Otero JE, Ying J, Duan X, Maruyama T, Rai MF, O'Keefe RJ, Mbalaviele G, et al.(2020). LDHA-mediated ROS generation in chondrocytes is a potential therapeutic target for osteoarthritis . Nature communications.

Adapala NS, Swarnkar G, Arra M, Shen J, Mbalaviele G, Ke K, Abu-Amer Y(2020). Inflammatory osteolysis is regulated by site-specific ISGylation of the scaffold protein NEMO . eLife.

Sul OJ, Kim JE, Ke K, Suh JH, Choi HS(2020). Atherogenic diet-induced bone loss is primarily due to increased osteoclastogenesis in mice . The Journal of nutritional biochemistry.

Ke K, Arra M, Abu-Amer Y(2019). Mechanisms Underlying Bone Loss Associated with Gut Inflammation . International journal of molecular sciences.

Ke K, Chen TH, Arra M, Mbalaviele G, Swarnkar G, Abu-Amer Y(2019). Attenuation of NF-κB in Intestinal Epithelial Cells Is Sufficient to Mitigate the Bone Loss Comorbidity of Experimental Mouse Colitis . Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research.

Sul OJ, Sung YB, Rajasekaran M, Ke K, Yu R, Back SH, Choi HS(2018). MicroRNA-155 induces autophagy in osteoclasts by targeting transforming growth factor β-activated kinase 1-binding protein 2 upon lipopolysaccharide stimulation . Bone.

Ke K, Sul OJ, Chung SW, Suh JH, Choi HS(2017). Lack of NOD2 attenuates ovariectomy-induced bone loss via inhibition of osteoclasts . The Journal of endocrinology.

Ke K, Sul OJ, Rajasekaran M, Choi HS(2015). MicroRNA-183 increases osteoclastogenesis by repressing heme oxygenase-1 . Bone.

Ke K, Safder AM, Sul OJ, Suh JH, Joe Y, Chung HT, Choi HS(2015). Cilostazol attenuates ovariectomy-induced bone loss by inhibiting osteoclastogenesis . PloS one.

Ke K, Safder MA, Sul OJ, Kim WK, Suh JH, Joe Y, Chung HT, Choi HS(2015). Hemeoxygenase-1 maintains bone mass via attenuating a redox imbalance in osteoclast . Molecular and cellular endocrinology.

Ke K, Sul OJ, Choi EK, Safdar AM, Kim ES, Choi HS(2014). Reactive oxygen species induce the association of SHP-1 with c-Src and the oxidation of both to enhance osteoclast survival . American journal of physiology. Endocrinology and metabolism.

Phan TV, Ke K, Sul OJ, Park YK, Kim KK, Cho YS, Chung HT, Choi HS(2014). Protection against ovariectomy-induced bone loss by tranilast . PloS one.

Van Phan T, Sul OJ, Ke K, Lee MH, Kim WK, Cho YS, Kim HJ, Kim SY, Chung HT, Choi HS(2013). Carbon monoxide protects against ovariectomy-induced bone loss by inhibiting osteoclastogenesis . Biochemical pharmacology.

Ke K, Sul OJ, Kim WK, Lee MH, Ko MS, Suh JH, Kim HJ, Kim SY, Park JW, Choi HS(2013). Overexpression of developmentally regulated GTP-binding protein-2 increases bone loss . American journal of physiology. Endocrinology and metabolism.

Ke K, Kim WK, Sul OJ, Phan VT, Lee MH, Kim HJ, Kim SY, Choi HS(2012). Elevation of fibrinogen due to loss of ovarian function enhances actin ring formation and leads to increased bone resorption . American journal of physiology. Endocrinology and metabolism.

Sul OJ, Ke K, Kim WK, Kim SH, Lee SC, Kim HJ, Kim SY, Suh JH, Choi HS(2012). Absence of MCP-1 leads to elevated bone mass via impaired actin ring formation . Journal of cellular physiology.

Kim WK, Ke K, Sul OJ, Kim HJ, Kim SH, Lee MH, Kim SY, Chung HT, Choi HS(2011). Curcumin protects against ovariectomy-induced bone loss and decreases osteoclastogenesis . Journal of cellular biochemistry.