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プロフィール詳細
Dr. Madeline S.に依頼
United States
Neuroscience expert with 8+ years of experience in cell and molecular biology research
プロフィール概要
専門分野
サービス
職務経験
Postdoctoral Fellow: Nestor and Dykxhoorn Lab
Hussman Institute
4月 2019 - 4月 2020
Postdoctoral Fellow: Torres and DiCicco-Bloom Lab
Rutgers University
2月 2018 - 12月 2018
PhD Candidate: DiCicco-Bloom Lab
Rutgers University
5月 2013 - 1月 2018
学歴
PhD | Neuroscience
Rutgers University, New Brunswick - United States
8月 2012 - 1月 2018
Bachelors in Science (Biochemistry)
Virginia Tech
8月 2008 - 5月 2012
認定資格
- 認定資格の詳細は未入力です。
出版物
JOURNAL ARTICLE
Madel Durens and Jonathan Nestor and Madeline Williams and Kevin Herold and Robert F. Niescier and Jason W. Lunden and Andre W. Phillips and Yu-Chih Lin and Derek M. Dykxhoorn and Michael W. Nestor(2020). High-throughput screening of human induced pluripotent stem cell-derived brain organoids . Journal of Neuroscience Methods. 335. Microsoft.AspNetCore.Mvc.Localization.LocalizedHtmlString 108627. Elsevier {BV}
Madeline Williams and Smrithi Prem and Xiaofeng Zhou and Paul Matteson and Percy Luk Yeung and Chi-Wei Lu and Zhiping Pang and Linda Brzustowicz and James H. Millonig and Emanuel Dicicco-Bloom(2018). Rapid Detection of Neurodevelopmental Phenotypes in Human Neural Precursor Cells (NPCs) . Journal of Visualized Experiments. (133). {MyJove} Corporation
Autism neural precursor cells from both idiopathic and CNV 16p11.2 deletion patients exhibit dysregulation of proliferation and mitogenic responses. Stem Cell Reports.
BOOK CHAPTER
Luka Turkalj and Monal Mehta and Paul Matteson and Smrithi Prem and Madeline Williams and Robert J. Connacher and Emanuel DiCicco-Bloom and James H. Millonig(2020). Using iPSC-Based Models to Understand the Signaling and Cellular Phenotypes in Idiopathic Autism and 16p11.2 Derived Neurons . Advances in Neurobiology. Microsoft.AspNetCore.Mvc.Localization.LocalizedHtmlString 79--107. Springer International Publishing
DISSERTATION THESIS
(2018). A tale of two families: neural precursor cells from idiopathic autism patients exhibit proliferation phenotypes.