プロフィール詳細

プロジェクトを作成

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Dr. Rajesh V.に依頼

India

Seasoned Molecular Cancer Biologist, experience in DNA damage, tumor models of cancer, aging, genome instability,

プロフィール概要

Trained Cancer biologist aim to deliver leading advancements in healthcare sector focusing on cancer biology, disease mechanism, drug discovery and molecular basis of drug resistance. I am particularly interested in molecular biology of cancer via dissect underlying molecular mechanism that leads to tumor onset, maintenance and development. I have experience of working with mouse and zebrafish model of cancer & Aging and genomic instability.

専門分野

• ★ Cell Aging
• ★ Drug Discovery
• ★ Cancer Research
• ☆ Molecular Biology
• ☆ Biomarkers
• ☆ Genetics
• ☆ Cell Biology
• ☆ Cell Signaling Pathways
• ☆ Skin Cancer

サービス

• Writing
• Research
• Consulting
• Data & AI
• Product Development

Writing Clinical Trial Documentation, Technical Writing, Copywriting, Newswriting

Research Market Research, User Research, Meta-Research, Feasibility Study, Fact Checking, Gap Analysis, Gray Literature Search, Systematic Literature Review, Secondary Data Collection

Consulting Scientific and Technical Consulting

Data & AI Statistical Analysis, Data Visualization

Product Development Formulation, Stability/Shelf Life Testing, Product Evaluation, Material Sourcing, Product Validation, Manufacturing, Concept Development, Prototyping

職務経験

Researcher

University of Minnesota

6月 2018 - 現在

Postdoctoral research associate

Umass Medical school

1月 2014 - 現在

Post doctoral research associate

Umass medical school

1月 2014 - 8月 2016

Research Fellow

National Institute of Imunology

6月 2006 - 7月 2008

学歴

Ph.D.

VIB, K.U. Leuven, Belgium

1月 2009 - 6月 2013

Ph.D. (Biomedical)

K.U. Leuven

1月 2009 - 6月 2013

認定資格

• 認定資格の詳細は未入力です。

出版物

JOURNAL ARTICLE

Potent inhibition of influenza virus replication with novel siRNA-chimeric-ribozyme constructs. Journal of general virology.

p53 attenuates AKT signaling by modulating membrane phospholipid composition. Onco target.

RNF4 is required for DNA double-strand break repair in vivo. Cell death and differentiation.

Cop1 constitutively regulates c-Jun protein stability and functions as a tumor suppressor in mice. Journal of clinical investigation.

Ligand-activated BMP signaling inhibits cell differentiation and death to promote melanoma. Journal of clinical investigation.

Nucleic acid-mediated cleavage of M1 gene of influenza A virus is significantly augmented by antisense molecules targeted to hybridize close to the cleavage site. Molecular biotechnology.

Reciprocal repression between P53 and TCTP. Nature Medicine.