プロフィール詳細

プロジェクトを作成

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Dr. Sumit S.に依頼

India

Academic Researcher | Expert in Preclinical Studies | Literature reviews

プロフィール概要

I am a PhD in Pharmacology, currently involved in development of nano formulation of a natural PDE-5 inhibitor as a management option of Heart failure.I have published more than 10 papers in peer review journals. I have a brief experience in medical writing (both clinical and preclinical) and academics. I am looking for a long term freelance opportunity.

専門分野

• ★ Pharmacy Education
• ☆ Clinical Pharmacy
• ☆ Cardiovascular Disease

サービス

• Writing
• Research
• Data & AI
• Product Development

Writing Clinical Trial Documentation, Technical Writing

Research Market Research, Systematic Literature Review

Product Development Formulation

職務経験

Research Associate

Jamia Hamdard

3月 2021 - 現在

学歴

PhD (Department of Pharmacology)

Jamia Hamdard

2月 2013 - 2月 2019

認定資格

• 認定資格の詳細は未入力です。

出版物

JOURNAL ARTICLE

(2019). Icariin attenuates isoproterenol-induced cardiac toxicity in Wistar rats via modulating cGMP level and NF-κB signaling cascade . Human & experimental toxicology.

(2019). Nerolidol attenuates cyclophosphamide-induced cardiac inflammation, apoptosis and fibrosis in Swiss Albino mice . European journal of pharmacology.

(2018). Molecular mechanism involved in cyclophosphamide-induced cardiotoxicity: Old drug with a new vision . Life sciences.

(2018). Suppression of isoproterenol-induced cardiotoxicity in rats by raspberry ketone via activation of peroxisome proliferator activated receptor-α . European journal of pharmacology.

(2018). Intranasally administered pitavastatin ameliorates pentylenetetrazol-induced neuroinflammation, oxidative stress and cognitive dysfunction . Life sciences.

(2017). Raspberry ketone protects against isoproterenol-induced myocardial infarction in rats . Life sciences.

CLINICAL UPDATES ON DRUG - INDUCED CARDIOTOXICITY. International Journal of Pharmaceutical Sciences and Research.

Prophylactic treatment with icariin prevents isoproterenol-induced myocardial oxidative stress via nuclear Factor-Like 2 activation. Pharmacognosy Magazine.